query	query_gene_id	query_protein_id	source_reference	hit_rank	swissprot_acc	swissprot_entry	database_section	target_gene_id	description	organism	homolog_gene_name	percent_identity	alignment_length	query_coverage	evalue	bitscore	confidence	evidence_type	source_db	reference_gene_id	reference_function_zh	reference_function_en	reference_papers_json
LXL04_009350	gene-LXL04_009350	KAL4584741.1	OH-1004	1	Q9SGU2	SAU71_ARATH	Swiss-Prot		Auxin-responsive protein SAUR71 OS=Arabidopsis thaliana OX=3702 GN=SAUR71 PE=2 SV=1	Arabidopsis thaliana	SAUR71	42.029	69	14.655	4.14e-11	63.2	weak_database_homology	BLASTP to reviewed UniProt Swiss-Prot	UniProt Swiss-Prot current_release 2026-06-10	At1g56150	在细胞膨胀、根分生组织模式形成和生长素运输调控中起作用。	Plays a role in the regulation of cell expansion, root meristem patterning and auxin transport.	[]
LXL04_009350	gene-LXL04_009350	KAL4584741.1	OH-1004	2	Q9LTV3	SAU72_ARATH	Swiss-Prot		Auxin-responsive protein SAUR72 OS=Arabidopsis thaliana OX=3702 GN=SAUR72 PE=1 SV=1	Arabidopsis thaliana	SAUR72	39.13	69	14.655	2.26e-09	58.9	weak_database_homology	BLASTP to reviewed UniProt Swiss-Prot	UniProt Swiss-Prot current_release 2026-06-10	At3g12830	介导生长素信号与质膜H⁺-ATP酶（AHA1、AHA2）之间的联系；通过抑制PP2C-D类磷酸酶，促进H⁺-ATP酶C端自抑制结构域的磷酸化及酶的活化，从而使质外体酸化，促进溶质和水吸收以及细胞扩张。	Provide a mechanistic link between auxin and plasma membrane H(+)-ATPases (PM H(+)-ATPases, e.g. AHA1 and AHA2), and triggers PM H(+)-ATPases activity by promoting phosphorylation of their C-terminal autoinhibitory domain as a result of PP2C-D subfamily of type 2C phosphatases inhibition, thus leading to the acidification of the apoplast and the facilitation of solutes and water uptake to drive cell expansion.	[]
LXL04_009350	gene-LXL04_009350	KAL4584741.1	OH-1004	3	O64538	SAU40_ARATH	Swiss-Prot		Auxin-responsive protein SAUR40 OS=Arabidopsis thaliana OX=3702 GN=SAUR40 PE=1 SV=1	Arabidopsis thaliana	SAUR40	39.394	66	14.009	6.33e-09	57.8	weak_database_homology	BLASTP to reviewed UniProt Swiss-Prot	UniProt Swiss-Prot current_release 2026-06-10	At1g79130	介导生长素信号与质膜H⁺-ATP酶（AHA1、AHA2）之间的联系；通过抑制PP2C-D类磷酸酶，促进H⁺-ATP酶C端自抑制结构域的磷酸化及酶的活化，从而使质外体酸化，促进溶质和水吸收以及细胞扩张。	Provide a mechanistic link between auxin and plasma membrane H(+)-ATPases (PM H(+)-ATPases, e.g. AHA1 and AHA2), and triggers PM H(+)-ATPases activity by promoting phosphorylation of their C-terminal autoinhibitory domain as a result of PP2C-D subfamily of type 2C phosphatases inhibition, thus leading to the acidification of the apoplast and the facilitation of solutes and water uptake to drive cell expansion.	[]
LXL04_009350	gene-LXL04_009350	KAL4584741.1	OH-1004	4	Q9SA49	SAU41_ARATH	Swiss-Prot		Auxin-responsive protein SAUR41 OS=Arabidopsis thaliana OX=3702 GN=SAUR41 PE=2 SV=1	Arabidopsis thaliana	SAUR41	37.333	75	15.948	1.36e-08	57.0	weak_database_homology	BLASTP to reviewed UniProt Swiss-Prot	UniProt Swiss-Prot current_release 2026-06-10	At1g16510	在细胞扩张、根分生组织模式和生长素运输的调节中发挥作用。	Plays a role in the regulation of cell expansion, root meristem patterning and auxin transport.	"[{""pmid"":""23396598"",""title"":""Tissue-specific expression of SMALL AUXIN UP RNA41 differentially regulates cell expansion and root meristem patterning in Arabidopsis."",""doi"":""10.1093/pcp/pct028"",""url"":""https://pubmed.ncbi.nlm.nih.gov/23396598/"",""year"":""2013"",""journal"":""Plant & cell physiology"",""authors"":""Kong Y, Zhu Y, Gao C, She W, Lin W, Chen Y, Han N, Bian H, Zhu M, Wang J."",""matched_aliases"":[""SAUR41""],""evidence"":""UniProt FUNCTION; UniProt/PubMed metadata and gene/species abstract checks; EuropePMC cross-check where available"",""metadata_sources"":[""UniProt"",""PubMed"",""EuropePMC""],""fulltext_reviewed"":false}]"
LXL04_009350	gene-LXL04_009350	KAL4584741.1	OH-1004	5	Q9ZUZ3	SAU32_ARATH	Swiss-Prot		Auxin-responsive protein SAUR32 OS=Arabidopsis thaliana OX=3702 GN=SAUR32 PE=2 SV=1	Arabidopsis thaliana	SAUR32	41.892	74	15.086	5.69e-08	54.7	weak_database_homology	BLASTP to reviewed UniProt Swiss-Prot	UniProt Swiss-Prot current_release 2026-06-10	At2g46690	可能在顶端钩发育中发挥作用（参考文献7）。	May play a role in the apical hook development (Ref.7).	[]